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¹ Canadian Cooperative Wildlife Health Centre, Department of Veterinary Pathology, University of Saskatchewan, 52 Campus Drive, Saskatoon, Saskatchewan S7N 5B4, Canada
² Foothills Model Forest, Box 6330, Hinton, Alberta T7V 1X6, Canada
³ Corresponding author (email: marc.cattet{at}usask.ca)

ABSTRACT:   We diagnosed exertional myopathy (EM) in a grizzly bear (Ursus arctos) that died approximately 10 days after capture by leghold snare in west-central Alberta, Canada, in June 2003. The diagnosis was based on history, post-capture movement data, gross necropsy, histopathology, and serum enzyme levels. We were unable to determine whether EM was the primary cause of death because autolysis precluded accurate evaluation of all tissues. Nevertheless, comparison of serum aspartate aminotransferase and creatine kinase concentrations and survival between the affected bear and other grizzly bears captured by leghold snare in the same research project suggests EM also occurred in other bears, but that it is not generally a cause of mortality. We propose, however, occurrence of nonfatal EM in grizzly bears after capture by leghold snare has potential implications for use of this capture method, including negative effects on wildlife welfare and research data.
  Key words:  Aspartate aminotransferase, AST, creatine kinase, CK, exertional myopathy, grizzly bear, leghold snare, Ursus arctos.